The "Bell's smile" is characterized by an asymmetry caused by paralysis of one side of the face.
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Bell's palsy

Bell's palsy (facial palsy) is characterised by facial drooping on the affected half, due to malfunction of the facial nerve (VII cranial nerve), which controls the muscles of the face. Named after Scottish anatomist Charles Bell, who first described it, Bell's palsy is the most common acute mononeuropathy (disease involving only one nerve), and is the most common cause of acute facial nerve paralysis. The paralysis is of the infranuclear/lower motor neuron type. Bell’s palsy affects about 40,000 people in the United States every year. It affects approximately 1 person in 65 during a lifetime. Until recently, its cause was unknown in most cases, but it has now been related to both Lyme disease and Herpes simplex. more...

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Symptoms

Additional symptoms that may accompany the condition are pain around the ear and loss of taste. In the great majority of patients, only one side of the face is affected. Detection of sensory loss, hearing loss, or ataxia during examination militates against the diagnosis of Bell's palsy and suggests the need for further evaluation.

Investigation

Clinicians should determine whether all branches of the facial nerve are involved, or whether the forehead muscles are spared. Since these receive innervation from both sides of the brain, the forehead can still be wrinkled by a patient whose facial palsy is caused by a problem in the brain rather than in the facial nerve itself.

Diagnosis

Bell's palsy is a diagnosis of exclusion; in many cases, no specific cause can be ascertained.

Pathology

It is supposed to be the result of inflammation of the facial nerve, which produces pressure on the nerve as it exits the skull within its bony canal. Patients with facial palsy for which an underlying cause can be readily found are not generally considered to have Bell's palsy per se. These underlying problems include tumor, meningitis, stroke, diabetes mellitus, head trauma and inflammatory diseases of the cranial nerves (sarcoidosis, brucellosis, etc). In these conditions, the neurologic findings are rarely restricted to the facial nerve. Babies can be born with Facial palsy, and they exhibit many of the same symptoms as people with Bell's palsy; this is often due to a traumatic birth which causes irrepairable damage to the facial nerve, i.e.acute facial nerve paralysis.

One disease that may be difficult to exclude in the differential diagnosis is involvement of the facial nerve in infections with the herpes zoster virus. The major differences in this condition are the presence of small blisters, or vesicles, of the external ear and hearing disturbances, but these findings may occasionally be lacking.

In recent years, two new suspects have been added to the possible causes of Bell's palsy. Lyme disease may produce the typical palsy, and may be easily diagnosed by looking for Lyme-specific antibodies in the blood. In endemic areas Lyme disease may be the most common cause of facial palsy. The subsequent observation of an increased incidence of antibodies to the Herpes simplex virus in patients with Bell's palsy has led many specialists to believe that this agent is the most likely underlying cause in areas where Lyme disease is uncommon.

Epidemiology

Bell's Palsy is three times more likely to strike pregnant women than non-pregnant women . It is also considered to be four times more likely to occur in diabetics than the general population, and it is more common in the elderly than children .

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Facial nerve hemangioma - Imaging Clinic - Bell's palsy
From Ear, Nose & Throat Journal, 11/1/03 by Enrique Palacios

Bell's palsy accounts for almost 50% of all cases of facial nerve paralysis. (1) Tumors are involved in only 5% of cases of facial paralysis, and the most common tumors are acoustic and facial neuromas. (2) Facial nerve hemangiomas account for only 0.7% of all temporal bone tumors. (1-3) Facial nerve hemangiomas arise from vascular plexuses distributed along the facial nerve paths in the geniculate ganglion region, in the mastoid segment of the facial nerve near the origin of the chorda tympani, and in the internal auditory canal around Scarpa's ganglion. (2)

Microscopically, facial nerve hemangiomas are seen as large vascular spaces lined with thin endothelium surrounded by thick walls filled with uniform fibrous tissue. (1,2) The characteristic honeycomb appearance seen on computed tomography (CT) (figure, A) is the result of erosion of the temporal bone and calcification of the collagen-containing walls between the vascular channels. The presence of bony channels, indistinct tumor margins, and intratumor bony spicules led to the term ossifying hemangiomas. (3)

[FIGURE A OMITTED]

High-resolution CT and enhanced magnetic resonance imaging (MRI) (figure, B) together are the most sensitive diagnostic modalities available to demonstrate facial nerve hemangiomas. (1)

[FIGURE B OMITTED]

Facial nerve hemangiomas and facial nerve neuromas yield similar clinical findings. Characteristically, severe nerve dysfunction is seen in patients with small hemangiomas; tinnitus and dizziness are seldom present. Conversely, symptomatic facial nerve neuromas are relatively large. Facial nerve hemangiomas and neuromas can be differentiated by their histologic appearance and by the presence or absence of the ossifying characteristics of facial nerve hemangiomas. (2) Facial nerve hemangiomas should also be distinguished from meningiomas, congenital cholesteatomas, metastatic carcinomas, and other vascular tumors that involve the temporal bone.

Surgical removal is the definitive treatment. In view of the extrinsic growth pattern of these tumors, surgical removal should be performed early to preserve facial nerve function. (4) Tumors in the geniculate ganglion region usually lead to an intense perineural reaction or direct nerve infiltration. Therefore, the opportunities to preserve an intact nerve following surgery are limited. Treatment by total excision and facial nerve grafting can provide a permanent cure and restore facial function. (2,5)

References

(1.) Salib RJ, Tziambazis E, McDermott AL. et al. The crucial role of imaging in detection of facial nerve haemangiomas. J Laryngol Otol 2001;115:510-13.

(2.) Escada P, Capucho C, Silva JM, el al. Cavernous haemangioma of the facial nerve. J Laryngol Otol 1997:111:858-61.

(3.) Friedman O. Neff BA, Willcox TO, et al. Temporal bone hemangiomas involving the facial nerve. Otol Neurotol 2002;23:760-6.

(4.) Alobid I, Gaston F, Morello A, et al. Cavernous haemangioma of the internal auditory canal. Acta Otolaryngol 2002:122:501-3.

(5.) Pulec JL. Facial nerve angioma. Ear Nose Throat J 1996:75: 225-38.

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